How To Deal With NEONATAL JAUNDICE

Prepaid by :-
DAVE ANAND G.
BAROT PANKAJ P.
SALVI MEHUL C.

As per KASYAP

neonatal jaundice symptoms :-

• eyes,nail,stool & urine seen yellowish
• anorexia,less activity

As per charak

Jaundice is a last stage of anemia
Its other symtopms :-

• yellowish skin
• Undigestion
• weakness
• anorexia

TYPES OF NEONATAL
JAUNDICE

• 1-Physiological
• 2-Obstructive
• 3-Infective
• 4-G6PD
• 5-Syphilitic
• 6-jaundice due to Hepatitis
• 7-Jaundice due to RH incompatibility

PHYSIOLOGICAL JAUNDICE

• Physiological jaundice is very comman in new born
• It’s occur on 3 or 4 days after birth
• It disappears gradually within 10 to 15 days

SYMPTOMS :-

• whole body is seen yellowish
• only sclera , palm of hand & leg are natural
• no changes color in stool & urine
• no deformity seen spleen & liver
• Bilirubin level is high in blood[normal value 1mg %]

ETIOLOGY :-

• The main cause of this jaundice is
• breakdown of R.B.C On birth time count of R.B.C. are 6.5mill/cmm, It decreases about 4.5mill/cmm in one week
• bilirubin level increases because of R.B.C break down
• neonatal liver is insufficiency of the liver enzyme glucoronyl transfarase
• So Neonatal liver is unable for metabolism of higher bilirubin
• so bilirubin goes into blood and that’s way skin become yellow

TREATMENT

• No specific treatment
• Only advice to parents for sun bath to child
• phototheropy

OBSTRUCTIVE JAUNDICE

• It’s called also congenital defect of bile
• duct or Immaturity of bile duct
• In this jaundice bile duct deformity since
• birth
• Some time in neonatal bile duct
• undeveloped or obstructed
• So bile is not passed in small intestine but
• goes direct in blood so skin become yellow

SYMPTOMS

• Skin , Sclera ,Stool, Urine are seen yellowish
• Nausea ,vomiting
• Liverenlarge

TREATMENT

• To remove obstruction of bile duct by surgery
• No drugs helpful in this case

INFECTIVE JAUNDICE

• It’s also called SEPTICEMIC JAUNDICE or HIGH MORTALITY
• It’s due to infection
• It’s spread by umbilical cord or other causes

SYMPTOMS

• Skin yellowish
• Nausea & vomiting
• Fever
• Hepatitis
• Some time we can’t decide infected area

TREATMENT

• To find infected area and remove it
• Light & liquid diet
• Give its Separate room
• Take all precaution for sterilization
• COMPLICATION :-
• still to the death due to the infection

G6PD DEFICIENCY JAUNDICE

• G6PD :- Glucose 6 phosphate dehydrogenaze
• G6PD is a enzyme which is essential for the maintaining the stability of R.B.C. membrane
• When deficiency of this enzyme in blood ,R. B.C is decreased before time thusjaundice become it is a heredity

SYPHILITIC JAUNDICE

• If the syphilis infection in parent‘s blood therefore the infection transfer in foetus by placenta
• Its called also congenital jaundice

JAUNDICE DUE TO HEPATITIS

• It arise in child into the 6 week after birth
• TRANSFER :- some times a mother suffering from hepatitis during pregnancy then infection transfer into foetus

SYMPTOMS :-

• hepato-spleenomegali

JAUNDICE DUE TO RH INCOMPATIBILITY

• It is very severe and dangerous
• It’s called also heamolitice jaundice
• first pregnancy :-
• mother :-Rh (-ve)
• father :- Rh (+ve)
• foetus :-Rh (+ve)
• That condition in mother there is no Rh(+ve) antibody so serviva
• second pregnancy :-
• mother :-Rh (-ve)
• father :- Rh (+ve)
• foetus :-Rh (+ve)
• So Produced Rh(+ve)antibody in mother blood breaks down foetus
• R.B.C. So Rh incompatibility happans in foetu

COMPLICATION

• If mother had an abortion or a miscarraige and second foetus have Rh + ve factor so this disease can be
• happened in foetus Generely it’s happed in foetus during pregnancy.AND also see after birth
• After birth when billirubine is more then there titer its enter brain cell and it effect on CNS .so child brain undeveloped or paralysis
• So it’s called central jaundice
• If that condition happened in prag. Foetus is death that’s called Hydrops fetalis
• IUFD = INTRACUTERAIN FOETAL DEATH

TREATMENT

• •AYURVEDA :-
  • -Bilvadidhrut(vagbhatt)
  • -amala,haldi,durva & gaumutra
  • -mandurbhasma , lohabhasmasuvarnamaxikbhasma arogyavardhini ,punarnavadi mandur
  • -liv -52
  • -livomin
  • -Hepatogard
  • -Livotreat

• •MORDEN :-
  • Remove its cause
  • 1)phenobarbitole :- increase liver enzyme activity
  • 2)Ager-Ager :-billirubine —->s.billirubine
  • 3)Photo theropy :-
  • blue light flurosent
  • morning sun light
  • PHOTOTHERAPY —–>billirubine ——>bilivedine
  • 4)Blood transfusion is necessary in hemolytic jaundice

DIAGNOSIS

• ST DAY :- Due to G6PD
  • Rh incompatibility
  • intrauterine infection
• 2nd & 3rd day :
  • physiological
• 4th to 7th day :-
  • septicemic
  • syphilitic jaundice
  • obstructive jaundice
• After 1 week :- septicemic
  • neonatal hepatitis

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