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How To Deal With NEONATAL JAUNDICE

Prepaid by :-
DAVE ANAND G.
BAROT PANKAJ P.
SALVI MEHUL C.

As per KASYAP

neonatal jaundice symptoms :-

  • eyes,nail,stool & urine seen yellowish
  • anorexia,less activity

As per charak

Jaundice is a last stage of anemia
Its other symtopms :-
  • yellowish skin
  • Undigestion
  • weakness
  • anorexia

TYPES OF NEONATAL
JAUNDICE

  • 1-Physiological
  • 2-Obstructive
  • 3-Infective
  • 4-G6PD
  • 5-Syphilitic
  • 6-jaundice due to Hepatitis
  • 7-Jaundice due to RH incompatibility

PHYSIOLOGICAL JAUNDICE

  • Physiological jaundice is very comman in new born
  • It’s occur on 3 or 4 days after birth
  • It disappears gradually within 10 to 15 days

SYMPTOMS :-

  • whole body is seen yellowish
  • only sclera , palm of hand & leg are natural
  • no changes color in stool & urine
  • no deformity seen spleen & liver
  • Bilirubin level is high in blood[normal value 1mg %]

ETIOLOGY :-

  • The main cause of this jaundice is
  • breakdown of R.B.C On birth time count of R.B.C. are 6.5mill/cmm, It decreases about 4.5mill/cmm in one week
  • bilirubin level increases because of R.B.C break down
  • neonatal liver is insufficiency of the liver enzyme glucoronyl transfarase
  • So Neonatal liver is unable for metabolism of higher bilirubin
  • so bilirubin goes into blood and that’s way skin become yellow

TREATMENT

  • No specific treatment
  • Only advice to parents for sun bath to child
  • phototheropy

OBSTRUCTIVE JAUNDICE

  • It’s called also congenital defect of bile
  • duct or Immaturity of bile duct
  • In this jaundice bile duct deformity since
  • birth
  • Some time in neonatal bile duct
  • undeveloped or obstructed
  • So bile is not passed in small intestine but
  • goes direct in blood so skin become yellow

SYMPTOMS

  • Skin , Sclera ,Stool, Urine are seen yellowish
  • Nausea ,vomiting
  • Liverenlarge

TREATMENT

  • To remove obstruction of bile duct by surgery
  • No drugs helpful in this case

INFECTIVE JAUNDICE

  • It’s also called SEPTICEMIC JAUNDICE or HIGH MORTALITY
  • It’s due to infection
  • It’s spread by umbilical cord or other causes

SYMPTOMS

  • Skin yellowish
  • Nausea & vomiting
  • Fever
  • Hepatitis
  • Some time we can’t decide infected area

TREATMENT

  • To find infected area and remove it
  • Light & liquid diet
  • Give its Separate room
  • Take all precaution for sterilization
  • COMPLICATION :-
  • still to the death due to the infection

G6PD DEFICIENCY JAUNDICE

  • G6PD :- Glucose 6 phosphate dehydrogenaze
  • G6PD is a enzyme which is essential for the maintaining the stability of R.B.C. membrane
  • When deficiency of this enzyme in blood ,R. B.C is decreased before time thusjaundice become it is a heredity

SYPHILITIC JAUNDICE

  • If the syphilis infection in parent‘s blood therefore the infection transfer in foetus by placenta
  • Its called also congenital jaundice

JAUNDICE DUE TO HEPATITIS

  • It arise in child into the 6 week after birth
  • TRANSFER :- some times a mother suffering from hepatitis during pregnancy then infection transfer into foetus

SYMPTOMS :-

  • hepato-spleenomegali

JAUNDICE DUE TO RH INCOMPATIBILITY

  • It is very severe and dangerous
  • It’s called also heamolitice jaundice
  • first pregnancy :-
  • mother :-Rh (-ve)
  • father :- Rh (+ve)
  • foetus :-Rh (+ve)
  • That condition in mother there is no Rh(+ve) antibody so serviva
  • second pregnancy :-
  • mother :-Rh (-ve)
  • father :- Rh (+ve)
  • foetus :-Rh (+ve)
  • So Produced Rh(+ve)antibody in mother blood breaks down foetus
  • R.B.C. So Rh incompatibility happans in foetu

COMPLICATION

  • If mother had an abortion or a miscarraige and second foetus have Rh + ve factor so this disease can be
  • happened in foetus Generely it’s happed in foetus during pregnancy.AND also see after birth
  • After birth when billirubine is more then there titer its enter brain cell and it effect on CNS .so child brain undeveloped or paralysis
  • So it’s called central jaundice
  • If that condition happened in prag. Foetus is death that’s called Hydrops fetalis
  • IUFD = INTRACUTERAIN FOETAL DEATH

TREATMENT

  • •AYURVEDA :-
    • -Bilvadidhrut(vagbhatt)
    • -amala,haldi,durva & gaumutra
    • -mandurbhasma , lohabhasmasuvarnamaxikbhasma arogyavardhini ,punarnavadi mandur
    • -liv -52
    • -livomin
    • -Hepatogard
    • -Livotreat
  • •MORDEN :-
    • Remove its cause
    • 1)phenobarbitole :- increase liver enzyme activity
    • 2)Ager-Ager :-billirubine —->s.billirubine
    • 3)Photo theropy :-
    • blue light flurosent
    • morning sun light
    • PHOTOTHERAPY —–>billirubine ——>bilivedine
    • 4)Blood transfusion is necessary in hemolytic jaundice

DIAGNOSIS

  • ST DAY :- Due to G6PD
    • Rh incompatibility
    • intrauterine infection
  • 2nd & 3rd day :
    • physiological
  • 4th to 7th day :-
    • septicemic
    • syphilitic jaundice
    • obstructive jaundice
  • After 1 week :- septicemic
    • neonatal hepatitis

What do you think?

Written by DD

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